New PDF release: Biological Monitoring: Heutige und künftige Möglichkeiten in

By Jürgen Angerer

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Es gibt Anhaltspunkte dafür, dass dieses Glutamin-Konjugat, das üblicherweise mit bis zu 70 % an der Ausscheidung der Butoxyessigsäure beteiligt ist, bei einzelnen Personen nicht oder nur in geringem Umfang gebildet wird. Als mögliche Konsequenz resultiert ein bei gleicher Exposition erhöhtes Risiko, eine Hämolyse zu entwickeln. Die Expositionskontrolle über die Bestimmung der Butoxyessigsäure, auch nach vorheriger Hydrolyse des Konjugats, ist ungeeignet, diese Risikopersonen zu identifizieren.

Sanderson, B. J. , Harris, E. L. : Glutathione S-transferase M1 (GSTM1) deficiency and lung cancer risk, Cancer Epidemiol. , 4, 589–594 (1995). -I. , 51, 5177–5180 (1991). -I. , 53, 2994–2999 (1993). , 44, 195–205 (1996). , Anttila, S. : Expression and polymorphism of glutathione S-transferase in human lungs: risk factors in smoking-related lung cancer, Carcinogenesis, 16, 707–711 (1995). , Auburtin, G. : Anti-benzo(a)pyrene diol-epoxide-DNA adduct levels in peripheral mononuclear cells from coke oven workers and the enhancing effect of smoking, Carcinogenesis, 16, 1373–1376 (1995).

3 Biological Monitoring of Arylamines and Nitroarenes preceding acid treatment. MDA and AcMDA were found in the urine of 84 % MDA-exposed workers and 78 % MDI-exposed workers. To release MDA and AcMDA from possible conjugates, urine was treated under strong acidic conditions. By this procedure the levels of MDA were found to be higher than the sums of MDA and AcMDA levels after base extraction alone. Urinary metabolites are indicators of recent exposure and it is not advisable to estimate an average daily dose from these values because there is a likelihood of over or under estimation (Cocker et al.

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